Antiheart

Then it can be presumed that antiheart serum, subsequently administered, causes yolk sac dysfunction of a higher degree, resulting in a distrubance in the metabolite transport essential to cardiogenesis.

Common antigenic determinants have been shown between streptococcal proteins and heart autoantigens, and T cell response to these determinants leads to the appearance of antiheart autoimmunity.

Titers of antiheart antibodies appeared to persist in patients with rheumatic recurrences.