Tubuloglomerular

Myogenic constriction and tubuloglomerular feedback mechanisms mediate renal autoregulation; however, intrinsic factors including nitric oxide and agonists for guanine nucleotide-binding protein (G protein)-coupled receptors (GPCRs), particularly angiotensin II via the renin-angiotensin system, regulate RBF by modulating renal vascular resistance (RVR) in pre- and post-glomerular arterioles.